
Dr
David A. Montgomery
Mr
John Reidy
Department of Surgery, Inverclyde Royal Hospital, Larkfield Road, Greenock, PA16 0XN
SMJ 2004 49(2): 71
Abstract
Giant
submucosal colonic lipomas are an uncommon cause of colonic symptoms. There is great debate in the literature as to how best to
deal with these. We present a
patient with a large colonic lipoma causing abdominal pain and altered bowel
habit. It was successfully treated
by open colostomy and resection.
Key
Words: submucosal, colonic, lipoma, mesenchymal, neoplasms, tumours.
Introduction
Colonic lipomas are benign tumours arising
from adipose connective tissue in the bowel wall. The first description is attributed to Bauer in 1757.
They are commonly described as a rare colonic tumour1,2, but
are in fact the most common benign mesenchymal tumour found in the colon3,
and the second most common benign colonic polyp after adenomatous polyps4.
Colonoscopy studies put the incidence at between 0.11% and 0.15%5,6,
while autopsy studies estimate prevalence at anywhere between 0.2% and 4.4% of
people7. Estimating
frequency is difficult, as most are small and asymptomatic8.
One review suggests an average size of 4.4 x 3.1 x 2.7 cm9.
Although uncommon, large colonic lipomas merit
attention, as they are often symptomatic and may be mistaken for malignancy9,10.
Case Report
We
report a case of a 73-year-old lady who presented to her GP with a long history
of altered bowel habit. She had
been suffering alternating periods of constipation and diarrhoea since
undergoing cholecystectomy for gallstone disease in 1994. In addition, in the
six months prior to visiting her GP she noticed increasing abdominal pain.
The pain was mainly in the right upper quadrant, radiating down the right
side. It was “gripping” in
nature, and episodes of pain were usually followed by passage of loose stool.
At the time of presentation to her GP, the patient was experiencing
episodes of pain every day and had suffered several episodes of faecal
incontinence. There had been no
mucus or blood passed per rectum, and no weight loss.
Colonoscopy
revealed a large mobile lesion in her colon arising from near the hepatic
flexure. Surface biopsies revealed normal colonic mucosa.
Barium enema showed the lesion to be a mobile mass 15 cm long by 4 cm in
diameter arising from a point of fixation on the lateral border of the upper
ascending colon and lying in the descending colon and caecum.
Barium flowed freely to the ceacum.
CT one month later revealed the lesion was now lying along the transverse
colon, filling 50% of the lumen. CT
density of the lesion suggested it was lipomatous.
A diagnosis of mobile submucosal colonic lipoma was made.
The lesion was not thought to be responsible for our patients symptoms,
and a period of observation was undertaken.
Worsening
of symptoms prompted a repeat CT 6 months later.
The appearances were unchanged. At
this point, she was referred for a surgical opinion.
She was taken to theatre on an elective basis and laparotomy was carried
out. Colotomy was performed and the
lesion was excised. Excision
resulted in a small defect in the muscle layer of the colon, and this was
oversown.

The
pathology report of the specimen confirmed it to be a submucosal lipoma, 8cm
long by 3cm in diameter. The
patient was followed up at clinic at six weeks and was pain free with normal
bowel motions.
Review
of the Literature
Autopsy
studies reveal colonic lipomas to be more common in women (56%) than men (44%)7,9.
They occur in older patients (average age 60)9.
70% are localized to the right hemi-colon; the caecum, ascending and
transverse colon in descending order of frequency10.
Men have a slight increased propensity towards left sided lesions10.
Pathologically, these tumours are well differentiated and arise from
adipose connective tissue in the bowel wall.
90% are submucosal in origin. The
rest are either subserosal (more common) or intramuscular mural9,10.
Colonic
lipomas are commonly small and asymptomatic.
Symptoms, when present, vary according to site of the lipoma.
Abdominal pain (23%) and rectal bleeding due to ulceration of overlying
mucosa (20%) are most common, with other symptoms such as anaemia, weight loss,
nausea, vomiting and abdominal distension reported less frequently9.
Abdominal pain can be attributed to several causes.
These include obstruction, particularly in the relatively rare rectal
lipomas, and intermittent intussusception, of which lipomata are the most
frequent cause in adults9. Other
dramatic presentations include perforation and appendicitis due to appendiceal
obstruction.
Diagnosis
can be difficult. However, pre-operative diagnosis is important as these lesions
present in a similar age group and with similar symptoms as do colonic
malignancies.
Pre-operative
diagnosis has been claimed by some authors to be possible in around 60% of
cases. It depends on a number of features being present.
The “squeeze” sign is seen radiographically and describes elongation
of a radio-opaque filling defect during peristalsis.
Lipomas can be directly visualized during colonoscopy and biopsies taken. Lipomas are covered by normal mucosa, and specimens will
reflect this. Normal mucosa on
biopsy does not rule out malignant connective tissue tumours, but is relatively
re-assuring for the lack of malignancy. Other
features on colonoscopy include the “tenting” sign, where the mucosa tents
over the lesion when grasped with forceps, the “cushion” sign, where
flattening of the lesion is followed by restoration of its shape on pressure
being removed, and the “naked fat” sign, where adipose tissue discharges
from the mucosal defect following biopsy7, 11-13.
CT and MRI may also have a role to play, but diagnosis is commonly
confirmed only after excision of the lesion.
The
vast majority of colonic lipomata are found incidentally.
Asymptomatic lesions should be left.
Larger lesions will often need resected, either due to symptoms or
uncertainty over diagnosis. There
is currently no consensus over how to proceed in this regard.
Several
studies report safe endoscopic removal of colonic lipomas14,15,16.
However, removal of lesions greater than 2 cm has been associated with a
higher risk of perforation14. Pfeil
et al, in a series of 7 patients, reported a 42.8% rate of perforation in
lipomas resected endoscopically16.
Risk factors for perforation were a broad base and a high volume on
histopathology. In all three cases
complicated by perforation, smooth muscle from the muscularis propria was
identified in the specimen, and one specimen contained serosa16.
Other authors report similar problems9, and muscularis was noted in
our specimen. The risk of
perforation has led numerous authors to advocate open surgery as the resection
method of choice7,11.
More
recently, several authors have proposed methods of ensuring safe endoscopic
removal colonic lipomas. Saline
injection to the base has been proposed17, and this is associated
with reduced risk of perforation by reducing the likelihood of ensnaring
underlying muscularis during resection. Kim
et al additionally utilised endoscopic ultrasound to evaluate the deep margins
of 4 colonic lipomas. In all four
cases, the lesion displayed endoscopic features typical of a lipoma, and
ultrasound revealed no extension into the underlying muscularis.
The base was then injected with either saline or a solution of 1:10,000
epinephrine (5-10ml) to raise the lesion. Endoscopic
removal was then carried out safely14.
Summary
Colonic
lipomas are not common, but do occur in clinical practice.
They are usually found incidentally and require no intervention. Occasionally, they cause symptoms and require resection.
Numerous diagnostic methods are available, but no method is definitive.
If doubt exists about the nature of a colonic lesion, resection should be
undertaken.
The
literature supports the contention that removal of lesions under 2cm in size by
colonoscopic means is safe. These
lesions are rarely symptomatic, however, and most would suggest that they can be
observed. Symptomatic lesions are
usually larger than 2cm. There is
no consensus over the correct management of larger lesions.
Various
techniques are described in the literature to maximise the safety of endoscopic
removal of colonic lipomas. However,
the majority of clinicians practicing in Britain will not have access to
endoscopic ultrasound to enable the full assessment of these lesions. Injection polypectomy does reduce the risk of perforation,
but will not eliminate it for the 10% of lipomas which arise from the muscularis.
It is the contention of this author that, In the absence of endoscopic
ultrasound, large symptomatic colonic lipomas would be more safely resected at
open surgery.
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